This beautiful breed is known for their intelligence and adaptability. Labradoodles are easy to train but should be exposed to formal training to reap lasting benefits. They are a loving companion, wonderful with children and thrive on family interaction. Their non-shedding coats (in multi-generational dogs) is a huge attraction to those who enjoy having their pets living alongside the family inside the home.
The range of colours is astounding and diverse, giving individuality to each dog. From the beautiful blacks whose coats shine in the light through to the reds, chocolates, creams, parti’s and so many other variations. Their coats require grooming (clipping) every 8 – 12 weeks, and they should be fully brushed every few days to avoid matts, which can become very painful.
Coats may be fleece, wool or hair. Hair coats are shedding coats, and mainly seen in the early generations. Fleece coats have a soft wavy appearance and wool coats have a curly appearance. Both fleece and wool coats are hypoallergenic.
There are three size ranges – miniature, medium and standard. The miniature height ranges from 35cm to 42cms with the weight range between 7kg to 13kg. The mediums range from 43 to 52 cm in height and 14 – 20 kg. The standard labradoodle will range from 53 - 63 cm with a weight range of 23 – 30kg.
All the parents of Aamberdell puppies have been health tested for the following:
Autosomal Hereditary Recessive Nephropathy
Centronuclear Myopathy (Labrador Retriever Type)
Cone-Rod Dystrophy 1 – PRA (cord 1)
Congenital Myasthenic Syndrome (Labrador Retriever Type)
Cystinuria (SLC3A1) Labrador Retriever Type
Elliptocytosis B-spectrin (Labrador Retriever/Poodle Type)
Exercise Induced Collapse (Retriever Type)
Gangliosidosis GM2 (Poodle Type)
Generalised PRA 1 (Golden Retriever Type)
Glodboid Cell Leukodystrophy/Krabbe’s Disease
Hereditary Nasal Parakeratosis/Dry Nose (Labrador Retriever Type)
Ivermectin Sensitivity MDR1 (Multi Drug Resistance)
Mild disproportionate Dwarfism (Labrador Type)
Mucopolysaccharidosis VI (Poodle Type)
Myotubular Myopathy X-Linked (Labrador Retriever Type)
Neonatal Encephalopathy (Poodle Type)
Phosphofructokinase Deficiency (Spaniel Type)
Progressive Rod Cone Degeneration (prcd) – PRA
Pyruvate Kinase Deficiency (Canine)
Pyruvate Kinase Deficiency (Labrador Type)
Skeletal Dysplasia 2 (Mild disproportionate Dwarfism)
Von Willebrand’s Disease Type 1
Hip and Elbow scores by PennHIP and the Orthopedic Foundation for Animals (OFA)
Even with the best of the Breeder's ability, sometimes Mother Nature may get it wrong, but if these tests are completed on the sire and dam, and followed up with good breeding practices, any health concerns in the puppy will be minimised.
This topic is such a contentious issue and one that we have spent a lot of time agonising over as to which way to go with our puppies. We want to do the right thing, and it seems the right thing is different for everyone. We've spent a year or so researching and talking to mentors and vets, all who come down on one side or the other. We’ve read many studies and could now convincingly argue both sides.
When it all boils down, it becomes a personal choice (for you and for us) after making an educated and thorough quest for all of the information. Our choice is to perform early-age desexing and here’s why we made that choice:
An argument against is that there are studies that report an incidence of urinary incontinence. These studies take a cross section of dogs (raw numbers) – both desexed early and later. They look at how many of each have incontinence and then come up with a ratio. The studies don’t have controls set and are not double blind. They are not from the same litter, the desexing was not performed by the same vet, they don’t have the same pedigree or history, so in fact, the results could be misleading. I’m not discounting that urinary incontinence could be caused by EAD, but here are some other feasible reasons for incontinence in dogs:
In essence there are so many variables that without controlled studies, no one can say definitively that EAD causes a great percentage of incontinence.
Another argument against is the possible occurrence of hip dysplasia – where the growth plates don’t close correctly. When we spoke to our vet who has had many years of dealing with labradoodle puppies and performing EAD (with no incontinence issues), she said that for small and medium dogs, EAD will not disadvantage them at all. What could cause hip dysplasia is:
Our dogs are small and medium. We have PennHip and OFA tests completed on all our breeding females and we won’t use a stud dog unless we have suitable PennHip, OFA and DNA tests for that dog.
There are many reasons why we think that EAD is the responsible decision:
We are not vets or scientists so we have to rely on the advice of those we trust (and not just Dr. Google) – our vet and institutions who have the best interest of the animals and the community at heart.
The one thing we keep coming back to is that the RSPCA (and other animal welfare groups) advocate early-age desexing. Now we know their agenda is to reduce the number of accidental litters and unwanted dogs having to be euthanised, but we would expect that they would not support EAD if it was in any way detrimental to an animal’s health. You may read what they have to say here and their report here.
We will be happy to change our stance on this if in the future the RSPCA and our vet come to the conclusion that EAD is not the way to go, but at this point in time, we are feeling a social conscience/duty towards not contributing to the 130,000 dogs euthanised in shelters across Australia each year. Our hearts break for them.
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